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Studies in the etiology of eclampsia
REVIEWS
Lawrence
:
Journal,
AND
Studies in the Etiology 1922,
xxv,
525
ABSTRACTS
of Eclampsia.
The
Pennsylvania
Medical
771.
Eclampsia is a generalized toxemia terminating in an anaphylactic reaction, the convulsion. The course of the disease i’s divided into three stages: mild, awere and convulsive. Low mentality, faulty personal hygiene and pre-ex&ing lesions of the eliminative systems seem to the author to be the factors det’ermining the extent and severity of the complex. Constipation, edema, headache snd albu-minuria are symptoms common to all stages of the toxemia. A period in which the body is oversaturated with foreign protein (chorin) is followed by acute pulmonary and cerebral edema and eonvulsionNs. As evidence of the anaphylactic nature of the convulsions the author calls attention to the favorable results’ from measures directed toward raising antibody production and decreasing the amount of foreign protein discharged into the system. Convulsions) follow fa.ilure of antibody production. Colonic irrigation, g,astrie lavage and morphine increase antibody formation. Fetal death, delivery and phlebotomy frequently control convulsions by ‘checking the production and distribution of fetal toxins. Repeated convulsions, pre-existing lesions of the liver and kidneys, anesthesia, too early administration of fo,od, cerebral stimulation by noise, light, etc., and failure to properly manage pulmonary edema are factors having an unfavorable influence on antibody formation. B:. W. SHUTTER.
Kark,
8. E.:
Journal,
Eclampsia: June 10, 1922,
Evolution
as
a
Causative
Factor.
Thze British
Medical
p. 912.
The author considers the, ch’anges which take place during pregnancy. He eonsiders the enlargement of the pituitary body during pregnancy as indicative of increase in its secretory function, and discusses a possible relationship b’etween hyperpituitarism and eclampsia. He dra#ws some analogy between acromegaly and eclampsia, and defines csertain symptoms as being due to changes in the anterior lobe and others due to changes in the posterior lobe of the hypophysis. He thinks that eclampsia is essentially a physiologic process overdoae’and is due to an exee’ss rather than a perversion of an essential product. F. L. ADAIR.
Wuth: xlviii,
The Eclampsia
Question.
Deutsche
medizinische
Woohenschrift,
192.2,
1339.
As a psychiatrist, Wuth interested himself in the eclampsia question and studied the blood of eclamptic patients in DGderlein’s clinic. Be was especially interested in Zangeme&ter ‘9 theory according to which eclampsia is caused by a retention of water in the tissues’ and incidentally in the blood. He did find ‘that in some cases there was a marked deficiency of serum albumin, which, however, was not The proportion of serum albumin, furthermore was not always in the oonstant. same ratio as the number of red blood cells which might be expected if the blood were simply diluted. Since convulsions of any kind have a tendency ‘to raise the quotient of serum albumin, Wuth feels that these findings do not contradict the theory of Zangemeister, even if the8y do not exactly prove it. He does not agree with Zengemeister when he thinks that the kidney function is not disturbed except that there is a retardation in the excretion of chloride. Like Caldwell and Lyle he found that retention ,of creatinin was the’ rule, while the percentage of urea wa,s constantly increased. He considers ‘this definite evidence of an impairment of the secretory function of the kidneys, but doe’s not thereby imply that kidney impairment is ‘the cause of eclampda, a question which, he feels, is yet to be settled. R. E: WOBUS.
Lawrence
:
Journal,
AND
Studies in the Etiology 1922,
xxv,
525
ABSTRACTS
of Eclampsia.
The
Pennsylvania
Medical
771.
Eclampsia is a generalized toxemia terminating in an anaphylactic reaction, the convulsion. The course of the disease i’s divided into three stages: mild, awere and convulsive. Low mentality, faulty personal hygiene and pre-ex&ing lesions of the eliminative systems seem to the author to be the factors det’ermining the extent and severity of the complex. Constipation, edema, headache snd albu-minuria are symptoms common to all stages of the toxemia. A period in which the body is oversaturated with foreign protein (chorin) is followed by acute pulmonary and cerebral edema and eonvulsionNs. As evidence of the anaphylactic nature of the convulsions the author calls attention to the favorable results’ from measures directed toward raising antibody production and decreasing the amount of foreign protein discharged into the system. Convulsions) follow fa.ilure of antibody production. Colonic irrigation, g,astrie lavage and morphine increase antibody formation. Fetal death, delivery and phlebotomy frequently control convulsions by ‘checking the production and distribution of fetal toxins. Repeated convulsions, pre-existing lesions of the liver and kidneys, anesthesia, too early administration of fo,od, cerebral stimulation by noise, light, etc., and failure to properly manage pulmonary edema are factors having an unfavorable influence on antibody formation. B:. W. SHUTTER.
Kark,
8. E.:
Journal,
Eclampsia: June 10, 1922,
Evolution
as
a
Causative
Factor.
Thze British
Medical
p. 912.
The author considers the, ch’anges which take place during pregnancy. He eonsiders the enlargement of the pituitary body during pregnancy as indicative of increase in its secretory function, and discusses a possible relationship b’etween hyperpituitarism and eclampsia. He dra#ws some analogy between acromegaly and eclampsia, and defines csertain symptoms as being due to changes in the anterior lobe and others due to changes in the posterior lobe of the hypophysis. He thinks that eclampsia is essentially a physiologic process overdoae’and is due to an exee’ss rather than a perversion of an essential product. F. L. ADAIR.
Wuth: xlviii,
The Eclampsia
Question.
Deutsche
medizinische
Woohenschrift,
192.2,
1339.
As a psychiatrist, Wuth interested himself in the eclampsia question and studied the blood of eclamptic patients in DGderlein’s clinic. Be was especially interested in Zangeme&ter ‘9 theory according to which eclampsia is caused by a retention of water in the tissues’ and incidentally in the blood. He did find ‘that in some cases there was a marked deficiency of serum albumin, which, however, was not The proportion of serum albumin, furthermore was not always in the oonstant. same ratio as the number of red blood cells which might be expected if the blood were simply diluted. Since convulsions of any kind have a tendency ‘to raise the quotient of serum albumin, Wuth feels that these findings do not contradict the theory of Zangemeister, even if the8y do not exactly prove it. He does not agree with Zengemeister when he thinks that the kidney function is not disturbed except that there is a retardation in the excretion of chloride. Like Caldwell and Lyle he found that retention ,of creatinin was the’ rule, while the percentage of urea wa,s constantly increased. He considers ‘this definite evidence of an impairment of the secretory function of the kidneys, but doe’s not thereby imply that kidney impairment is ‘the cause of eclampda, a question which, he feels, is yet to be settled. R. E: WOBUS.