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The role of rT3 in metabolism in obesity treatment
86 icated neuro-endocrine biological system for the sole purpose to induce feeding behaviour. A name is proposed for this system and some research are reviewed how it functions physiologically and also pathologically. It is argued that a proper descriptive name for this system has relevance for the treatment and understanding of obesity and further research into it. It is proposed that by far the most eating behaviours are normal physiological phenomena and that only a very small percentage can be regarded as pathophagia’s. http://dx.doi.org/10.1016/j.orcp.2014.10.156 176 The role of GH in obesity Hendrik Rensburg The Rensburg Clinic, Nedlands, Australia Hormonal factors in obesity and ageing are discussed with special reference to practical applications. The main focus will be on the role of GH and IGF-1 in catabiosis (medical name for ageing) and obesity. The origional name for GH, instead of the name ‘‘Growth Hormone’’ is proposed to be used instead of the name GH, to enhance the understanding of its functions in adults, since the present name appears to be flawed within the framework of current knowledge. (The name ‘‘obesity’’ is also seriously flawed, but this can be deliberated on during another talk. Various alternatives are proposed, especially one contender] The adrenopause, menopause, somatopause and viropause are known physiological, albeit pathological consequences of ageing. A new concept is proposed namely uvenipause to enhance the understanding and treatment of catabiosis. The extensive interactions of GH with other hormones are discussed. The role of GH in the treatment of obesity is examined and it is suggested that GH and IGF-1 are under utilized in the treatment of obesity. However, it is also not used correctly. GH is not a standalone hormone and needs various other helper hormones and life style changes to perform properly in the treatment of obesity and without the latter it is argued that it can be an expensive waste of money and resources. GH controversies like side effects, endocrine suppression and cancer fears are analysed. http://dx.doi.org/10.1016/j.orcp.2014.10.157
M. Pickford 177 The role of rT3 in metabolism in obesity treatment Hendrik Rensburg The Rensburg Clinic, Nedlands, Australia The traditional TSH and at times a T4 are the accepted tests to determine thyroid function. However, in the case of Obesity, Insulin resistance and Leptin disturbances a patient often have a normal TSH and T4 according to the laboratory ranges, but still have symptoms of hypothyroidism. The body seems to adapt to weight loss by conserving energy. The metabolism does not know the difference between a diet and a famine, even though the person intellectually knows what they are doing, the metabolism is still in the stone age, even though we live in the space age. One of the ways its seems to do this is by causing a hypo-metabolic state by decreasing the conversion of T4 into T3 and the latter also sgets blocked at receptor level. Cells in the pituitary act different than cells in the rest of the body due to Deiodinases. Reverse T3 plays a crucial role here. Some patients find it almost impossible to keep their lost weight off as a result. With the appropriate treatment the rT3 can be decreased and the fT3:rT3 ratio can be increased leading to a more normal metabolic state with resultant increases in success rates in the treatment of obesity. (18953) http://dx.doi.org/10.1016/j.orcp.2014.10.158 89 Systematic review of incidental physical activity community interventions: Results and contribution to government strategy Rebecca C. Reynolds 1,∗ , Stephen McKenzie 2 , Steven Allender 3 , Kirsty Brown 2 , Chad Foulkes 2 1 UNSW
Australia, Sydney, NSW, Australia 2 City of Greater Geelong Council, Geelong, VIC, Australia 3 WHO Collaborating Centre for Obesity Prevention, Deakin University, Geelong, VIC, Australia Background and significance: Increasing incidental physical activity (PA) such as active transport has substantial public health potential.
M. Pickford 177 The role of rT3 in metabolism in obesity treatment Hendrik Rensburg The Rensburg Clinic, Nedlands, Australia The traditional TSH and at times a T4 are the accepted tests to determine thyroid function. However, in the case of Obesity, Insulin resistance and Leptin disturbances a patient often have a normal TSH and T4 according to the laboratory ranges, but still have symptoms of hypothyroidism. The body seems to adapt to weight loss by conserving energy. The metabolism does not know the difference between a diet and a famine, even though the person intellectually knows what they are doing, the metabolism is still in the stone age, even though we live in the space age. One of the ways its seems to do this is by causing a hypo-metabolic state by decreasing the conversion of T4 into T3 and the latter also sgets blocked at receptor level. Cells in the pituitary act different than cells in the rest of the body due to Deiodinases. Reverse T3 plays a crucial role here. Some patients find it almost impossible to keep their lost weight off as a result. With the appropriate treatment the rT3 can be decreased and the fT3:rT3 ratio can be increased leading to a more normal metabolic state with resultant increases in success rates in the treatment of obesity. (18953) http://dx.doi.org/10.1016/j.orcp.2014.10.158 89 Systematic review of incidental physical activity community interventions: Results and contribution to government strategy Rebecca C. Reynolds 1,∗ , Stephen McKenzie 2 , Steven Allender 3 , Kirsty Brown 2 , Chad Foulkes 2 1 UNSW
Australia, Sydney, NSW, Australia 2 City of Greater Geelong Council, Geelong, VIC, Australia 3 WHO Collaborating Centre for Obesity Prevention, Deakin University, Geelong, VIC, Australia Background and significance: Increasing incidental physical activity (PA) such as active transport has substantial public health potential.