BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS ARTICLE NOS.
225, 695 (1996)
1232–1235
ERRATA
Volume 215, Number 3 (1995), in the article ‘‘pH Dependent Alterations of Monoepoxides and Monochlorohydrins of Linoleic Acid, and Their Existence in Vivo,’’ by Hirotaro Iwase, Kimie Takahashi, Takehiko Takatori, Takao Shimizu, Kazunori Aono, Yoshihiro Yamada, Kimiharu Iwadate, and Masataka Nagao, pages 945–951: On page 945, in the author line the name of the second author is incorrect as printed. The name should be Toshie Takahashi instead of Kimie Takahashi. Volume 219, Number 2 (1996), in the article ‘‘Superoxide Anion Reduces the Ability of Myeloperoxidase to Damage Lipids,’’ by Hirotaro Iwase, Takehiko Takatori, Masataka Nagao, Kimiharu Iwadate, Megumi Takahashi, Makoto Nakajima, Kimie Takahashi, and Takao Shimizu, pages 625–632: On page 625, in the author line the name of the seventh author is incorrect as printed. The name should be Toshie Takahashi instead of Kimie Takahashi. Volume 222, Number 2 (1996), in the article ‘‘Glutathione-Linked Thiol Peroxidase Activity of Human Serum Albumin: A Possible Antioxidant Role of Serum Albumin in Blood Plasma,’’ by Mee-Kyung Cha and Il-Han Kim, pages 619–625: On page 624, Fig. 7, pathway labels I and II were inadvertently switched. The legend was correct as printed. For the reader’s convenience, the corrected Fig. 7 and its legend are reprinted here.
FIG. 7. Mechanism for the reduction of H2O2 by HSA with glutathione (GSH) as an in vivo electron donor.
Volume 224, Number 2 (1996), in the article ‘‘A Novel Donor Splice Site Mutation in the Glycogen Debranching Enzyme Gene Is Associated with Glycogen Storage Disease Type III,’’ by Minoru Okubo, Yoshiko Aoyama, and Toshio Murase, pages 493–499: On page 495, in the legend to Fig. 1, line 4 should read ‘‘. . . M: pBR322-Hae III . . .’’ instead of ‘‘. . . M: fX174-Hae III. . . .’’ For the reader’s convenience, the corrected legend is printed below. FIG. 1. (A) Agarose gel electrophoresis of amplified fragment B of debrancher cDNA. The patient had a single fragment shorter than the normal one. M: l-Hind III marker; N: normal control; P: patient. (B) Nucleotide sequence of fragment B from the patient. 124 base pairs (bp) of exon M (5*-TACATGTT---AATAAGAG) were missing. (C) Polyacrylamide gel electrophoresis of an amplified cDNA fragment encompassing the exon M. M: pBR322-Hae III marker; N: normal control; P: patient.
695 Copyright q 1996 by Academic Press, Inc. All rights of reproduction in any form reserved.
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