Volume 52, July. 1967
No.
cine ical
1
From
the Clinics
College, York, 1941.
14
F.
AND
IN
Union
Med-
Publishers,
TIJONO,
K.,
B. C.
KAMMINGA,
3.:
Inc.,
on
CORONARY 25
New
WILLEBRANDT,
of
“Influence different forms
nutrition, individuality and stress
STRESS
of Peiping
Interscience
J. 3.,
GROEN,
A.
EMOTIONAL
26
of
blood cholesterol: results of an ex9 months duration in 60 normal Proc. First Congress Dietetics, Voeding 10:1959. 15 BARROW, J. G., QUINLAN, C. B., EDMANDS, R. E. AND RoDlLosso, P. T.: Prevalence of Atherosclerotic Complications in Trap pist and Benedictine Monks, Presented at 34th Scientific Sessions American Heart Assn., Bal Harbour, Florida. 16 LAPICCIRELLA, V., LAPICCIRELLA, R., ABBONI, F. AND LIOTTA, S.: “Enquete clinique, biologique et cardiographique parmi les tribus nomades de Ia Somalie qui se nournissent seulement de lait,” Bulletin de l’Organisation Mondiale de Ia Sante, 27:681, 1962. 17 MANN, G. V., SHAFFER, R. D. AND RICH, A.: “Physical fitness and immunity to heart disease in Masai,” Lancet, 2: 1308, 1965. 18 STOUT, C., MORROW, 3., BRANDT, E. N.,JR. AND WOLF, S.: “Unusually low incidence of death from myocardial infarction; study of an Italian-American community in Pennsylvania,” JAMA, 188:845, 1964. L9 LOWN, B. AND SnARE, F. 3.: “Atherosclerosis, infarction and nutrition,” Circulation, 20: 161, 1959. 20 RATCLIFFE, H. L., YERASIMIDES, T. H. AND ELLIOTT, G. A.: “Changes in the character and location of arterial lesions in mammals and birds in the Philadelphia Zoological Garden,” Circulation, 21:730, 1960. 21 RIKLI, A. E. AND OTHERS “Prevalence of coronary heart disease,” U. S. Public Health Survey, Public Health Reports, Aug. 1960. 22 MYASNIKOV, A. L.: “Influence of some factors on development of experimental cholesterol atherosclerosis,” Circulation, 17:99, 1958. 23 TSIBEKMAKHER, T. D.: Quoted in reference periment volunteers,”
of
22.
H. clotting
UHLEY,
24
lipids, rats
mt.
N.
exposed
J.
Am.
Physiol.,
AND
FRIEDMAN,
and
to
a
coronary particular
197:396,
AFFECTION In
the
adults
104
author
majority
tant third
affection
cases
with
the
1959.
THE
form
of the heart. and objective
as
alteration of the results
diagnostic
and
cardiogram
fourth
were
the
dyspnea data:
of
IN
of
nature
x-ray
of
heart
represented
by
medium
I.
AND HUMPHREVS, R. 3.: myocandial epinephrine-sympathin
W.
RAAB, action
concentration
“Atherosclerosis-a commentary,” No.2: Part 2:47, 1959.
upon
“Drug
and heart rate: (nitroglycerine, pniscol, di ben amine hydrochloJ. Pharm. Exp. Ther., 89:64, 1947. W., CHAPLIN, J. P. AND BAJUSZ, E.:
papavenine, ride), 30
RAAB,
“Myocardial
31
necroses produced
rats and in wild rats stresses,” Proc. Soc. 665, 1964. GROOVER, M. E., JR.,
3. J.
LIN,
dial
infarction
409,
32
in the atherosclerosis,
DREYFUSS,
35
and
uric
under
mt.
at STILL,
ed
viscosity ported April
AND
F.
choleserol Arch.
34
J.
in
at 26,
SHARNOFF,
CZACZKES,
acid
stress
Med.,
of
R.: baboon
J.
without
Angiology,
J.
of
L., HAG“Myocar-
E.
healthy
14:
W.:
“Blood medical AMA
examination,
1959.
AND HEIFFER, M. H.: “Blood response to various stimuli,” reFed. Amer. Soc. for Exper. Bio., 1958. J. G.: “Stress-caused platelet rise to post-op. clots,” reported in Med. meeting of Amer. Soc. Clin. Pa-
“Life, A.
stress and Res. Nerv.
Association of Heart write: Island,
RHEUMATISM
Dr. New
IN
bodily Ment.
Pamphlet: Attack,” 1966. Russek, York.
study
rheumatism of the heart
176
disDis., “ReHart
ADULTS
Phonocardiographic
primary
domesticated
W.
please Staten
fluctuations.
an
103:708,
called key News from thologists. WOLFF, H. G.: ease: formulation,” 29:1059, 1950. American Heart duce Your Risk
with
Kenya
1963.
students
33
SELJESKOG, HITCHCOCK, C.
AND
demonstrable
in
by sensory and emotional Exper. Rio. Med., 116:
In
enabled the a weakening,
patients
revealment
Importhe phono-
Kuaopov,
of
sounds,
signs of myocardlal affection heart sounds which on the
1960. H.: Proc., 18:
PAGE,
at the apex splitting prolongation of the duration of the first sound, systolic and mesodiatolic murmurs. In ECG investigation. myocardial involvement is manifested by a prolongation of the electric systole, disturbance and deceleration of intraatrial conduction, impairment of intraventricular conduction, pathologic change of the T wave and S-T segment, rarely prolongation of the atrioventnicular conduction.
involvement
investigation.
6:1,
PRIMARY
carditis, in the
9
C. G., FRIEDMAN, M. AND BYERS, S. 0.: Effect of chronic hypothalmic stimulation upon cholesterol-induced atherosclerosis in the rabbit,” J. Clin. Invest., 39:1963, 1960. BOODONOFF, M. D., E5TES, E. H., Ja. AND HARLAN, W.: “Psychophysiologic studies of fat metabolism,” read before the Fortieth Annual Session, Amen. College of Physicians, Chicago, April 21, 1959. STEINBERG, D. AND SHAFRIR, E.: “Cortisone held ‘vital’ to lipid rise in stress,” Med. News,
For reprints, Boulevard,
stress,”
HEART
on exertion, enlargement
29
DISEASE
GUNN,
Fed.
in
rheumatoid of the heart prevalent
28
36
“Blood
myocardium. Lesion of the endocardium at later periods of the disease. The of myocarditis could be established on of the patients’ complaints of pain in
region cardia
heart, well
revealed of
of the found sence basis
OF
primary
with
M.: atherosclerosis
27
HEART
was prethe the
tachythe
are
frequency
as
matism
A. in
I.;
“Affection
of
adults.”
Soviet
Med.,
the
heart 29:i4.
in 1966.
primary
rheu-