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Cardiac hypertrophy and coronary arteriosclerosis in hypertension
SELECTED
543
ABSTRACTS
Kahn, Joseph R., and Ingraham, Edgar S., Jr.: Cardiac Hypertrophy Arteriosclerosis in Hypertension. Arch. Path. 31: 373, 1941.
and Coronary
Within the limitations of this sort of statistical study, the results suggest that arteriosclerosis of the coronary arteries may play a part in l.he genesis of cardiac hypertrophy in patients with prolonged hypertension. AUTHORS.
Katzenstein, Rolf, and Murphy, James Peter: Acute Sclerosing With Renal Changes. Arch. Int. Med. 67: 579, 1941.
Vascular
Disease
An unusual case of vascular disease, occurring in a 22.year-old woman, is presented. Sclerosing changes in small arteries, without thrombosis, characterized the vascular Clinically, the illness lesions. The cause of the condition is entirely unknown. lesions (with subsequent simulated bilateral renal cortical necrosis. Arteriolar cerebral injury) were noted, and their possible pathogenesis is discussed. AUTHORS.
Burrett, John B., and Scherf, David: The Clinical Importance of Small Intracutaneous Veins in the Human Chest. Am. J. M. SC. 201: 399, 1941. Ectasia of small intracutaneous veins located in the neighborhood of the pleural sinuses is described. An examination of 385 patients revealed that the appearance of these veins is not the result of pulmonary, pleural, or cardiac disease. They are found in healthy people. Their incidence increases with age. An explanation of their developmental mechanism is offered. AUTHORS.
Steinbrocker, Extremities
Otto, and Samuels, Saul S.: The Arterial Circulation of the Lower in Chronic Arthritis. J. Lab. & Clin. Med. 26: 974, 1941.
In a clinical investigation of rheumatoid arthritis, 65.9 per cent of the patients showed abnormalities, usually vasomotor disturbances, of the arterial circulation of the lower extremities; in osteoarthritis 35.2 per cent of the cases presented similar findings. Three rheumatoid patients, or 6.3 per cent had simultaneous arteriosclerotic occlusive disease; two with osteoarthritis, or 2.8 per cent, presented evidence of arteriosclerosis obliterans. Study of those patients with arthritis presenting arterial abnormalities showed no direct, consistent relationship between the location of the vascular signs and the site of the arthritic process. The evidence suggests that the vasomotor disturbances in chronic rheumatoid arthritis and related conditions may be attributed to some systemic reaction probably acting through the sympathetic nervous system, although in some cases a local irritative reflex vasospastic mechanism in the painful arthritic extremity cannot be ruled out. In a control group of eighty-six patients with advanced organic arterial disease twenty-eight, or 32 per cent, offered a history, physical signs, or roentgen changes of rheumatic or arthritic involvement at some time, These findings approximate the incidence of such ailments in similar age groups of persons without arterial disturbances and contribute clinical evidence that pronounced arterial disease alone does not usually produce arthritic signs and symptoms. AUTHORS.
543
ABSTRACTS
Kahn, Joseph R., and Ingraham, Edgar S., Jr.: Cardiac Hypertrophy Arteriosclerosis in Hypertension. Arch. Path. 31: 373, 1941.
and Coronary
Within the limitations of this sort of statistical study, the results suggest that arteriosclerosis of the coronary arteries may play a part in l.he genesis of cardiac hypertrophy in patients with prolonged hypertension. AUTHORS.
Katzenstein, Rolf, and Murphy, James Peter: Acute Sclerosing With Renal Changes. Arch. Int. Med. 67: 579, 1941.
Vascular
Disease
An unusual case of vascular disease, occurring in a 22.year-old woman, is presented. Sclerosing changes in small arteries, without thrombosis, characterized the vascular Clinically, the illness lesions. The cause of the condition is entirely unknown. lesions (with subsequent simulated bilateral renal cortical necrosis. Arteriolar cerebral injury) were noted, and their possible pathogenesis is discussed. AUTHORS.
Burrett, John B., and Scherf, David: The Clinical Importance of Small Intracutaneous Veins in the Human Chest. Am. J. M. SC. 201: 399, 1941. Ectasia of small intracutaneous veins located in the neighborhood of the pleural sinuses is described. An examination of 385 patients revealed that the appearance of these veins is not the result of pulmonary, pleural, or cardiac disease. They are found in healthy people. Their incidence increases with age. An explanation of their developmental mechanism is offered. AUTHORS.
Steinbrocker, Extremities
Otto, and Samuels, Saul S.: The Arterial Circulation of the Lower in Chronic Arthritis. J. Lab. & Clin. Med. 26: 974, 1941.
In a clinical investigation of rheumatoid arthritis, 65.9 per cent of the patients showed abnormalities, usually vasomotor disturbances, of the arterial circulation of the lower extremities; in osteoarthritis 35.2 per cent of the cases presented similar findings. Three rheumatoid patients, or 6.3 per cent had simultaneous arteriosclerotic occlusive disease; two with osteoarthritis, or 2.8 per cent, presented evidence of arteriosclerosis obliterans. Study of those patients with arthritis presenting arterial abnormalities showed no direct, consistent relationship between the location of the vascular signs and the site of the arthritic process. The evidence suggests that the vasomotor disturbances in chronic rheumatoid arthritis and related conditions may be attributed to some systemic reaction probably acting through the sympathetic nervous system, although in some cases a local irritative reflex vasospastic mechanism in the painful arthritic extremity cannot be ruled out. In a control group of eighty-six patients with advanced organic arterial disease twenty-eight, or 32 per cent, offered a history, physical signs, or roentgen changes of rheumatic or arthritic involvement at some time, These findings approximate the incidence of such ailments in similar age groups of persons without arterial disturbances and contribute clinical evidence that pronounced arterial disease alone does not usually produce arthritic signs and symptoms. AUTHORS.