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Corrigendum to: “Autophagic and lysosomal reactions to stress in the hepatopancreas of blue mussels”
Aquatic Toxicology 86 (2008) 457
Corrigendum
Corrigendum to: “Autophagic and lysosomal reactions to stress in the hepatopancreas of blue mussels” Michael N. Moore a,∗ , Aldo Viarengo b , Peter Donkin a , Anthony J.S. Hawkins a b
a Plymouth Marine Laboratory, Prospect Place, The Hoe, Plymouth PL1 3DH, UK Department of Environment and Life Science, University of Piemonte Orientale, Alessandria, Italy
The author deeply regrets that an error occurred in the original printing of the above paper. The previously published micrograph in Fig. 6A was erroneously showing the effect of 3-methylcholanthrene on digestive cell lysosomes and not fluoranthene as stated. Also, the micrographs in Fig. 6C–E relate to the phenanthrene treatment and not fluoranthene as previously stated.
Fig. 6. Frozen tissue sections of hepatopancreas showing the effects of the PAHs fluoranthene and phenanthrene on digestive cells: (A) fluoranthene treated cells showing characteristic PAH-induced swollen autolysosomes (arrows) reacted for the lysosomal marker enzyme -N-acetylhexosaminidase; (B) pale blue intra-lysosomal fluorescence (arrows) due to accumulated fluoranthene; (C) phenanthrene induced crescentic accumulation of neutral lipid (oil red-O) in swollen autolysosomes (arrows); (D) control digestive cells (from the phenanthrene experiment) showing representative normal phospholipid-negative lysosomes (L) and residual bodies (RB, tertiary lysosomes) reacting for phospholipid; (E) phenanthrene-treated digestive cells showing swollen phospholipid-positive autolysosomes (AL) and residual bodies (RB, tertiary lysosomes) reacting for phospholipid. Arrows in A–C indicate representative autophagolysosomes (autolysosomes). Scale bars = 10 m. (For interpretation of the references to color in this figure legend, the reader is referred to the web version of the article.)
∗
DOI of original article:10.1016/j.aquatox.2007.06.007. Corresponding author. Tel.: +44 1752 633120; fax: +44 1752 633 101. E-mail address: [email protected] (M.N. Moore).
0166-445X/$ – see front matter © 2007 Elsevier B.V. All rights reserved. doi:10.1016/j.aquatox.2007.12.007
Corrigendum
Corrigendum to: “Autophagic and lysosomal reactions to stress in the hepatopancreas of blue mussels” Michael N. Moore a,∗ , Aldo Viarengo b , Peter Donkin a , Anthony J.S. Hawkins a b
a Plymouth Marine Laboratory, Prospect Place, The Hoe, Plymouth PL1 3DH, UK Department of Environment and Life Science, University of Piemonte Orientale, Alessandria, Italy
The author deeply regrets that an error occurred in the original printing of the above paper. The previously published micrograph in Fig. 6A was erroneously showing the effect of 3-methylcholanthrene on digestive cell lysosomes and not fluoranthene as stated. Also, the micrographs in Fig. 6C–E relate to the phenanthrene treatment and not fluoranthene as previously stated.
Fig. 6. Frozen tissue sections of hepatopancreas showing the effects of the PAHs fluoranthene and phenanthrene on digestive cells: (A) fluoranthene treated cells showing characteristic PAH-induced swollen autolysosomes (arrows) reacted for the lysosomal marker enzyme -N-acetylhexosaminidase; (B) pale blue intra-lysosomal fluorescence (arrows) due to accumulated fluoranthene; (C) phenanthrene induced crescentic accumulation of neutral lipid (oil red-O) in swollen autolysosomes (arrows); (D) control digestive cells (from the phenanthrene experiment) showing representative normal phospholipid-negative lysosomes (L) and residual bodies (RB, tertiary lysosomes) reacting for phospholipid; (E) phenanthrene-treated digestive cells showing swollen phospholipid-positive autolysosomes (AL) and residual bodies (RB, tertiary lysosomes) reacting for phospholipid. Arrows in A–C indicate representative autophagolysosomes (autolysosomes). Scale bars = 10 m. (For interpretation of the references to color in this figure legend, the reader is referred to the web version of the article.)
∗
DOI of original article:10.1016/j.aquatox.2007.06.007. Corresponding author. Tel.: +44 1752 633120; fax: +44 1752 633 101. E-mail address: [email protected] (M.N. Moore).
0166-445X/$ – see front matter © 2007 Elsevier B.V. All rights reserved. doi:10.1016/j.aquatox.2007.12.007