- Email: [email protected]
Does a primary hypertension of the lesser circuit exist? (Socalled primary pulmonary sclerosis.)
smEcTEDABSTRACTS
755
The persistence of action among the aglycones is slight; that is, they are all rapidly eliminated from the circulation. This is particularly true with digitoxigenin in contrast with digitoxin. Digitoxigenin caused a brief initial stimulation as manifested by convulsions, followed by marked depression of the central nervous system in cats and frogs. Digitoxin has no such action in corresponding doses. AUTHORS.
Kienle, F. : Clinical and Electrocardiographic Wall Infarct. Ztschr. f. Kreislaufforsch. An injured skier came to clinical evidence supported by indicated myocardial infarction.
the the
Observations 30:
674,
on Traumatic
Posterior
1935.
clinic with severe cardiac roentgen and electrocardiographic
insufficiency, and examination KATZ.
Staemmler, M. : Does a Primary Hypertension Called Primary Pulmonary Sclerosis.) Arch.
of the Lesser Circuit f. Kreislaufforsch.
The various anatomic forms of primary pulmonary the result of a primary hypertension of the arterioles The first change is hypertrophy of the media of the The other anatomic changes are medium sized arteries.
Exist?
(So-
3: 125, 1938.
sclerosis are considered to be of the pulmonary circulation. arterioles and elastica of the secondary. KATZ.
Kunkel, Paul, Stead, Eugene A., Jr,, and Weiss, Soma: BIood Flow and Vasomotor Reactions in the Hand, Forearm, Foot, and Calf in Response to Physical and Chemical Stimuli. J. Clin. Investigation 18: 225, 1939. Following strong sensory stimuli, such as pinching the skin until pain is caused, or a sudden, loud noise, the forearm and calf respond in three ways: (1) by a decrease in volume after a latent period of from 3 to 9 seconds; (2) by an increase in volume after a latent period usually not exceeding 2 seconds; (3) by a biphasic response with first an increase in volume with a short latent period usually not exceeding 2 seconds, and subsequently a decrease in volume. Reasons are stated for the belief that the decrease in volume is the result of active reflex vasoconstriction, while the increase in volume is the result of a transient increase in cardiac output and a passive distention of the vascular bed. The sensitivity of vasomotor responses varies considerably in different persons. Variations in the same individual have been observed from time to time, The vessels of the upper part of the body are more sensitive to physical, chemical, and nervous stimuli than those of the lower part of the body. The blood flow induced by local heat of 43” C. is greater in the hand and foot than in the forearm and calf even when the mass of muscle present in the forearm and calf is disregarded and when the blood flow is recorded as cubic centimeters per minute per 100 sq. cm. of surface area. Local heat of 43” C. produces nearly complete vasodilatation in the skin, but only relatively slight vasodilatation in the underlying muscles. Exercise is a very effective stimulus for producing vasodilatation in the muscles, but not in the skin. The ability of the muscle vessels to dilate in response to exercise is not appreciably influenced by varying the external temperature between 32” and 43” C. Pitressin, 1 C.C. intramuscularly, causes as great a decrease in blood flow in the hand and the foot at 43” C. as does epinephrine. At 43” C. it causes a moderate decrease in flow in the forearm and calf. Neither epinephrine nor pitressin interferes with the dilatation of the muscle vessels in response to exercise or arterial occlusion.
755
The persistence of action among the aglycones is slight; that is, they are all rapidly eliminated from the circulation. This is particularly true with digitoxigenin in contrast with digitoxin. Digitoxigenin caused a brief initial stimulation as manifested by convulsions, followed by marked depression of the central nervous system in cats and frogs. Digitoxin has no such action in corresponding doses. AUTHORS.
Kienle, F. : Clinical and Electrocardiographic Wall Infarct. Ztschr. f. Kreislaufforsch. An injured skier came to clinical evidence supported by indicated myocardial infarction.
the the
Observations 30:
674,
on Traumatic
Posterior
1935.
clinic with severe cardiac roentgen and electrocardiographic
insufficiency, and examination KATZ.
Staemmler, M. : Does a Primary Hypertension Called Primary Pulmonary Sclerosis.) Arch.
of the Lesser Circuit f. Kreislaufforsch.
The various anatomic forms of primary pulmonary the result of a primary hypertension of the arterioles The first change is hypertrophy of the media of the The other anatomic changes are medium sized arteries.
Exist?
(So-
3: 125, 1938.
sclerosis are considered to be of the pulmonary circulation. arterioles and elastica of the secondary. KATZ.
Kunkel, Paul, Stead, Eugene A., Jr,, and Weiss, Soma: BIood Flow and Vasomotor Reactions in the Hand, Forearm, Foot, and Calf in Response to Physical and Chemical Stimuli. J. Clin. Investigation 18: 225, 1939. Following strong sensory stimuli, such as pinching the skin until pain is caused, or a sudden, loud noise, the forearm and calf respond in three ways: (1) by a decrease in volume after a latent period of from 3 to 9 seconds; (2) by an increase in volume after a latent period usually not exceeding 2 seconds; (3) by a biphasic response with first an increase in volume with a short latent period usually not exceeding 2 seconds, and subsequently a decrease in volume. Reasons are stated for the belief that the decrease in volume is the result of active reflex vasoconstriction, while the increase in volume is the result of a transient increase in cardiac output and a passive distention of the vascular bed. The sensitivity of vasomotor responses varies considerably in different persons. Variations in the same individual have been observed from time to time, The vessels of the upper part of the body are more sensitive to physical, chemical, and nervous stimuli than those of the lower part of the body. The blood flow induced by local heat of 43” C. is greater in the hand and foot than in the forearm and calf even when the mass of muscle present in the forearm and calf is disregarded and when the blood flow is recorded as cubic centimeters per minute per 100 sq. cm. of surface area. Local heat of 43” C. produces nearly complete vasodilatation in the skin, but only relatively slight vasodilatation in the underlying muscles. Exercise is a very effective stimulus for producing vasodilatation in the muscles, but not in the skin. The ability of the muscle vessels to dilate in response to exercise is not appreciably influenced by varying the external temperature between 32” and 43” C. Pitressin, 1 C.C. intramuscularly, causes as great a decrease in blood flow in the hand and the foot at 43” C. as does epinephrine. At 43” C. it causes a moderate decrease in flow in the forearm and calf. Neither epinephrine nor pitressin interferes with the dilatation of the muscle vessels in response to exercise or arterial occlusion.