inflammation. In particular, HDL controls theflammatory in response in
genes related to inflammation and monocyte activation[29] . However, the in fluence of chronic low HDL on the ability of monocytes to differentiate toward M2 macrophages was not yet investigated in humans. We previously reported that circulating blood cells maintain a donor-related phenotype upon in vitroculture[30] . Thus, peripheral blood monon985.7H09-1.p913[5.7H0(33e38ph)-12h3Bb33e38phd1v4(per)-1d1v4(pf3B1)1v4(pf.g35(b)7.3v4.2547Tca813e38ph)-8.962b1rBt0t3Gc1k1.8fb13ob