Ue.rpite the lbrulongetl Ilyt~otc~llsiol,, rh:tng<~ an11 in the hematoc>rit. readings is not $1 riking.
;in(ni1t(t
iTi the
of
~)I:w~II:~
L)rol~~il~::
Parallelism seems to exist Iwt.wern the fall in I~10011 pressure nfter trauma (0 the nervous system an(l development ot’ nngiotonin refractoriness. After the s.vntry the injections of gum drome has developed, elevation ol’ the \,lood pressure acacia solution or whole hItrot dots not restore the responsiveness to angiotonin. ilngiotonin refractoriness Sul)rarenalectorIlrrhage.
and hypotension also or nephrectom~does not
develol~ prevent
after marked its :Ippearance.
hemor-
~~I:TIIOI:.
Logue, R. B.:
Dissecting
Aneurysm
of the Aorta.
A study of dissecting aneurysm of diagnosis was made in ten of the twelve
Am.
.l. Jr.
SC.
206:
the aorta is presented. An cases olwwwl ant1 conlirnit~d
.5-I, 1942.
ante-mortem on nrcropsy.
Isolated instances of dissecting aneurysm complicating calcareous aortic stenoA new sign of this disease sis and syphilitic aortic insufficiency are presented. is described, consisting of a bruit and thrill over the femoral artery. A history of previous hypertension, or an ele\-ation the time of admission, was noted in eleven of the twelve Tamponade was the cause of death cavity, uremia, coronary occlusion, causes of death.
in seven cases. ant1 ventricular
of the cases. Hemorrhage fil)rillation
I~lood
pressure
into were
at
the pleural the other AUTIIOR.
English, Arch.
J. P.. and Willius,
I?. A.:
Int.
1943.
Med.
71: 594,
Hemorrhagic
Lesions
of the
Coronary
Arteries.
Hemorrhagic lesions were observed in the wails of the coronary arteries in 54 of 135 hearts (40 per cent) and were directly or indirectly related to acute occlusion of the coronary artery in 40. The lesions were characterized by hemorrhage associated with the presence of large lipoicl-containing cells, proliferative intimal changes, and organization. Smaller: less active lesions usually were found adjacent to calcified plaques. The intimal changes that coexisted with the hemorrhage appeared to represent the primary factor in the pathologic condition: the hemorrhage was secondary. It does not seem logic:tl. moreover, that hemorrhage in itself can have protlucdell the effects ol)srt\-etl.
Medial Hypertrophy Graef, I.: Path. 19: 12 t, 1943.
of the Renal Arterioles
in Pregnancy.
Am.
.I.
A case is reported of chronic unilateral hematuria in a colored female, Zl years old, who was cured by nephrectonlg during the fifth month of pregnancy. The removed kidney revealed unusual, massive, medial hyperplasia and hypertroph> of the arterioles. There was accompanying intimal fihromuscular hyperplasia of the interlobular branches of the renal artery. Focal fresh hemorrhagic erosions were found in the pelvis and ureter. The medial hypertrophy could not be accounted for 1,~ any known or demonstrable injury. A hypothetical explanation is offered, based on the theory that hormonal activity during pregnancy might he responsil)le for changes in the arteriolar smooth muscle comparable to those found in the grayid uterus. ATTTHOI:.