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Incidence of cardiac diseases
The development of rheumatic activity \vlth a toxin-producing strain of hemolytie immune response to this infection.
appears to depend nut ~niy- spun :nket:o~, strept.ococcus, but also upon the host’s ~kT.Yl’HOR.
Goburn, A. F., and Pauli, R. H.: Studies on the Imune Xesponse of the Rheumatic Subject and Its Relationship to Activity of the Rheumatic Process. VI. The Significance of the Rise of Antistreptolysin Level in the Development of Rheumatic Activity. J. Clin. Investigation 14: 769, 1935. The median of the antistreptolysin determinations on 176 individuals in good health was, 71 units. This is somewhat higher than the natural human level of 50 units. The median titer developed in acute rheumatism was 500 units, and the geometric mean 490 units. In m03t, instances the titer returned to approximately natural level within a perio,d of one year. The onset of acute rheumatism coincided with a sharp rise in antistreptolysin titer. R,heumatic patients infected with hemolytic streptococcus who escaped recrudescence showed little or no change in antistreptolysin titer, The relation of the immune response of the host to the development of rheumatic activity is discussed. AUTHOR. Goburn, A. F., and Panli, Ruth H.: Studies on the Immune Response of the Rheumatic Subject and Its Relationship to Activity of the Rheumatic Process. VII. Splenectomy in Relation to the Development of Rheumatic Activity. J. Clin. Investigation 14: 783, 1935. Splenectomy did not permanently modify eit.her the immune response or the character of the rheumatic recrudescence. Nine out of twenty apparently quiescent rheumatic subjects developed recrudescences as a direct sequel to splencctomy. All of those nine individuals had elevated antistreptolysin titers at the time of operation, and none of them showed any increase in titer during or after the recrudescence. This shows that following operative manipulation of antibody-producing tissue an exacerbation of symptoms may develop in the during subsiding rheumatism, absence of further rise in antistreptolysin titer. AUTHOR. Moore,
A. 6.:
Incidence
of Cardiac
Diseases.
J. Indiana
RI. A.
28:
419,
1935.
It was found that 77.7 per cent of the cardiovascular abnormalities were hypertensive group. The author believes the increased nervous tension of is responsible for the constantly increasing cases in this group. The stmly that individuals with hypertension have a little longer span of life than in the valvular or nonhypertensive group. Primary occurrences of cases in the valvular group, especially in the earlier findings, are probably due to intercurrent infections. Palpable arteriosclerosis comes established fire years earlier in the hypertensive group than in the in practically the same hypertensive group, but following t.his, it is increased for both groups.
in the today shows those
Lewis, Thomas: The Ergot Poisoning.
Under
Manner in Which Necrosis Clin. Se. 2: 43, 1935,
Arises
in the
Fowl’s
comb
age benonratio
The poison ergotoxine constricts the arteries of the comb; this constriction is one that cannot be released by relaxation of vasomotor tone or by local warming. The constriction is maintained if ergot injections are repeated day by day, but it is insufficient to stop the blood tlow through the comb, which continues a little
appears to depend nut ~niy- spun :nket:o~, strept.ococcus, but also upon the host’s ~kT.Yl’HOR.
Goburn, A. F., and Pauli, R. H.: Studies on the Imune Xesponse of the Rheumatic Subject and Its Relationship to Activity of the Rheumatic Process. VI. The Significance of the Rise of Antistreptolysin Level in the Development of Rheumatic Activity. J. Clin. Investigation 14: 769, 1935. The median of the antistreptolysin determinations on 176 individuals in good health was, 71 units. This is somewhat higher than the natural human level of 50 units. The median titer developed in acute rheumatism was 500 units, and the geometric mean 490 units. In m03t, instances the titer returned to approximately natural level within a perio,d of one year. The onset of acute rheumatism coincided with a sharp rise in antistreptolysin titer. R,heumatic patients infected with hemolytic streptococcus who escaped recrudescence showed little or no change in antistreptolysin titer, The relation of the immune response of the host to the development of rheumatic activity is discussed. AUTHOR. Goburn, A. F., and Panli, Ruth H.: Studies on the Immune Response of the Rheumatic Subject and Its Relationship to Activity of the Rheumatic Process. VII. Splenectomy in Relation to the Development of Rheumatic Activity. J. Clin. Investigation 14: 783, 1935. Splenectomy did not permanently modify eit.her the immune response or the character of the rheumatic recrudescence. Nine out of twenty apparently quiescent rheumatic subjects developed recrudescences as a direct sequel to splencctomy. All of those nine individuals had elevated antistreptolysin titers at the time of operation, and none of them showed any increase in titer during or after the recrudescence. This shows that following operative manipulation of antibody-producing tissue an exacerbation of symptoms may develop in the during subsiding rheumatism, absence of further rise in antistreptolysin titer. AUTHOR. Moore,
A. 6.:
Incidence
of Cardiac
Diseases.
J. Indiana
RI. A.
28:
419,
1935.
It was found that 77.7 per cent of the cardiovascular abnormalities were hypertensive group. The author believes the increased nervous tension of is responsible for the constantly increasing cases in this group. The stmly that individuals with hypertension have a little longer span of life than in the valvular or nonhypertensive group. Primary occurrences of cases in the valvular group, especially in the earlier findings, are probably due to intercurrent infections. Palpable arteriosclerosis comes established fire years earlier in the hypertensive group than in the in practically the same hypertensive group, but following t.his, it is increased for both groups.
in the today shows those
Lewis, Thomas: The Ergot Poisoning.
Under
Manner in Which Necrosis Clin. Se. 2: 43, 1935,
Arises
in the
Fowl’s
comb
age benonratio
The poison ergotoxine constricts the arteries of the comb; this constriction is one that cannot be released by relaxation of vasomotor tone or by local warming. The constriction is maintained if ergot injections are repeated day by day, but it is insufficient to stop the blood tlow through the comb, which continues a little