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Ion flux and contractility
SESSION II Monday 27th July (14.00 - 17.30) ION FLUX AND CONTRACTILITY Chair: D. Noble (Oxford, U.K.)
MOLECULAR FUNCTION OF THE Na/Ca EXCHANGER: GUINEA-PIG, RAT AND HUMAN. W.J. Lederer, P. Kofuji, D. Schulze, R. Hadley, R. Kieval, M.S. Kirby and E. Niggli, Departments of Physiology, Pharmacology and Microbiology, University of Maryland, 660, W. Redwood Street, Baltimore, MD 21201, USA and Department of Physiology, University of Bern, Bern, Switzerland. DEPOLARIZATION OF RAT CARDIAC MYOCYTES INDUCES Ca ENTRY ON Na/Ca EXCHANGE WHICH TRIGGERS INTRACELLULAR Ca RELEASE AND CONTRACTION. AJ. Levi, P. Brooksby and J.C. Hancox, Department of Physiology, School of Medical Sciences, University of Bristol, Bristol BSS lTD, UK RESTITUTION AND POTENTIATION STUDIED BY VOLTAGE CLAMP. M.I.M. Noble, P. Arlock, B. Wohlfart, Departments of Zoophysiology and Clinical Physiology, University of Lund, Sweden and Academic Unit of Cardiovascular Medicine, Charing Cross and Westminster Medical School, London SWlP 2AR, UK Ca’+ GATING OF CARDIAC RYANODINE RECEPTOR: IS Ca2+ CHANNEL A PRMLEGED ROUTE? M. Morad, J.S.K. Sham, L. Cleemann, Department of Physiology, University of Pennsylvania, Philadelphia, PA 191046085, USA EFFICACY OF BRIEF CazC CURRENTS IN TRIGGERING Ca2+ RELEASE DEPENDS ON SR Ca” LOAD (GUINEA-PIG VENTRICULAR MYOCYTES). G. Isenberg, S. Han, M-F. Wendt-Gallitelli. Departments of Physiology, University of Cologne and University of Tubingen, Germany. AMINO ACIDS AND INTRACELLULAR SODIUM IN CARDIAC MUSCLE R.A. Chaoman, BHF Research Group, Department of Physiology, School of Veterinary Science, University of Bristol, Bristol BSl 5LS, UK
MOLECULAR FUNCTION OF THE Na/Ca EXCHANGER: GUINEA-PIG, RAT AND HUMAN. W.J. Lederer, P. Kofuji, D. Schulze, R. Hadley, R. Kieval, M.S. Kirby and E. Niggli, Departments of Physiology, Pharmacology and Microbiology, University of Maryland, 660, W. Redwood Street, Baltimore, MD 21201, USA and Department of Physiology, University of Bern, Bern, Switzerland. DEPOLARIZATION OF RAT CARDIAC MYOCYTES INDUCES Ca ENTRY ON Na/Ca EXCHANGE WHICH TRIGGERS INTRACELLULAR Ca RELEASE AND CONTRACTION. AJ. Levi, P. Brooksby and J.C. Hancox, Department of Physiology, School of Medical Sciences, University of Bristol, Bristol BSS lTD, UK RESTITUTION AND POTENTIATION STUDIED BY VOLTAGE CLAMP. M.I.M. Noble, P. Arlock, B. Wohlfart, Departments of Zoophysiology and Clinical Physiology, University of Lund, Sweden and Academic Unit of Cardiovascular Medicine, Charing Cross and Westminster Medical School, London SWlP 2AR, UK Ca’+ GATING OF CARDIAC RYANODINE RECEPTOR: IS Ca2+ CHANNEL A PRMLEGED ROUTE? M. Morad, J.S.K. Sham, L. Cleemann, Department of Physiology, University of Pennsylvania, Philadelphia, PA 191046085, USA EFFICACY OF BRIEF CazC CURRENTS IN TRIGGERING Ca2+ RELEASE DEPENDS ON SR Ca” LOAD (GUINEA-PIG VENTRICULAR MYOCYTES). G. Isenberg, S. Han, M-F. Wendt-Gallitelli. Departments of Physiology, University of Cologne and University of Tubingen, Germany. AMINO ACIDS AND INTRACELLULAR SODIUM IN CARDIAC MUSCLE R.A. Chaoman, BHF Research Group, Department of Physiology, School of Veterinary Science, University of Bristol, Bristol BSl 5LS, UK