s5
mapping (IBM).
Up to 16 EIX
time using the first unit. epoch selection spectral
derivations
Different
can also be performed density
function
evolution of EEG therefore
is estimated
clinical
produce the image on the TBM on a two-dimensional
are located.
levels of quantiration
The
power
The
time can
domain analyses are and application.
To
of the head. Parameters
to estimate
The
permits
with type I; 80% with type 2; 100% with type 3. In conclusion BAliI’s were
best
predictor
population
the
of
comatose
differential
sensitivity
groups.
The
of
unfavourable
patients.
of each modality
combination
of
outcome
Our
findings
in the
in the individual
alterations
a
actiological
in multimodality
evoked
response seems to suggest an individual profile for each specific patient. An
assessment
combination
of
the
significance
prognostic
of alterations
is currently
of
the
individual
in progress.
like EEG
index arc mapped
the values at points where no
mapping
values are exhibited
the
design
of
systems
IBNGTERM
EEG AND
(LTM) IN EPILEPSY
ECG MONITORIN<:
AND EPISODIC PHENOMENA.
using 14
with
D. Bar&n,
Z. Sabaka, I. Brezny and I>. Buranova
different
as well as rapid system upgrade by adding new units for
(Centre
for Stroke and CVD.
Bratislava,
Czechoslovakia)
tasks. Many factors can influence the amount
EI,ECTRODIA<:NOSTIC
STUDIES
IS MYASTHENIA
GRAVIS.
clinical
the degree of alertness during the day and
and quality of sleep during the night.
and
subclinical
symptoms
of
‘lhe occurrence
short-term
disturbances
consciousness can be enhanced or suppressed depending Z.G.
general
also document
unit, a basic 20x27 grid is superimposed
and assigning a colour pattern of 2x2 pixels. The
approach
configurations
in real-
scale and
unit.
rate and power proportionality
using a spatial interpolation
different
unit.
on the SA
interpretation
projection
rhythm, theta/alpha
modular
on the DIXG
Both time and frequency
to facilitate
electrodes
amplitude
spectra. using compressed spectral arrays (CSA),
be displayed.
performed
can be monitored
montages,
Bo,gcrbaldo, S. Lim-LopeT,
L.D.
Renales.
CM.
Danguilan,
M.C.
Perc7.
vigilance.
Typical
arrhythmia,
examples
of such symptoms
College of Medicine.
Manila,
Philippines)
and monitor I:EG normal
Serial repetitive
nctve stimulation
of the ulnar or lateral
peroneal
facial nerves was performed
at one to several weeks intervals
patients
clinically diagnosed
as having myasthenia
frequent
type of response (62.5%)
to the 5th response. There A minimum decrement
The
most
of the 2nd
were 5 other patterns of response obtained.
of 11.8% was significant at the 95% level. After
4-minutes
nerve
compound
muscle action potentials
stimulation,
show no decrement
gravis.
was an early decrement
or
on 69
the
decrement
or
increased.
or increment
increment
of
the
There was a tendency to
when the patient
became symptom-
of
on the state of
are epilepsy,
cardiac
sudden drop of blood pressure and others.
To establish such interaction, (University
of
it is necessary to record day and night,
activity
and IXG
as well as blood pressure during the
life-style.
The patient material
described consists of 387 patients divided into
the following 3 groups: 1 - a clear diagnosis of epilepsy; 2 - suspected epilepsy, 3 -short-term
disturbances of consciousness of unknown origin.
The aim of the study was to answer the following epilepsy in patients with unclear or probable what is the frequency of epileptic LEG
1 - can
and how does this compare with the frequency
of clinical seizures; 3 I
and others).
(cardiac arrhythmia,
The
following
methods
free and when they went into stable remission (no symptoms and no
were used: 1 - 3 activating techniques: a) the sudden discontinuation
medication).
anti-epileptic
drugs, b) sleep deprivation,
2 - cassette recording
of EEG
2 -
activity during a 24 hour period
what is the frequency of other episodic phenomena sudden drop of blood pressure
questions:
epilepsy be confirmed;
c) pharmacological
of
activation:
and EC<;.
In the cases with epilepsy, only 18.5% specific epileptic I;l:G changes PROGNOSTIC
VALUE
POTENTIALS
OF
IN PATIENTS
.MULTIMODALITY
WITH
SEVERE
COMA
EVOKED
were found, the rest were normal
OF DIFFER:NG
AETIOLOGIES. A. Burelli, R. Proietti.
A. Arcangeli,
R. Calimici,
M. Valente
and G.1,.
Gigli
or non-specific.
‘Ihe rate of specific
episodic findings increased after sleep deprivation
in 24%, but the rate
of such findings in LTM
was 42.0%
was 72hrs.
between
Correlation
(P > 0.05). clinical
Ihe
seizures
time of recording and episodic
discharges shows significant valuable results in I,TM. observed
interesting
results
regarding
the
time-course
of epileptic
discharges over 24 hours and propose a diagnostic therapeutic (Universita
Cattolica
Multimodality (GCS
del S. Cuore,
evoked potentials
6) admitted
haemorrhage,
to our
(CII)
(14)
Rome, Italy)
1111G
The authors have program
which can improve validity in the diagnosis of episodic phenomena.
were studied in 46 comatose patients
ICU
for head injury
ischemic-anoxic
(III)
(19),
encephalopathy
cerebral
(IAE)
(7)
NEUROPHYSIOLOGICAL ADMINISTRATION
CHANGES
FOLLOWIN<;
CIIRONIC
OF CARRAMAZEPINE.
ischemic stroke (S) (4), neoplasm (2). Evoked responses obtained in all patients included brainstem auditory auditory
potentials
(MLAEPs)
Our purpose was to compare the entire
series of patients
Recordings were performed
potentials (BAEPs),
and somatosensory
Prognosis was evaluated
(1983)
were
the event. utilized
percentage of mortality with type I was (III L4E 80%). CtI
The percentage
85.7: L4E
100%
significance,
independent
classes (1 =N20
bilaterally
3= N20 not identifiable
R. Rocchi. S. Rossi, R. Cioni, S. Passer0 and
C. Paradiso in
groups.
on the basis
Morphological
classes
to classify BAEPs. 11.5%; S 0.0%;
MLAEPs
among types III, demonstrated
of aetiology. identifiable;
bilaterally).
66.3:
IV and V was
a poor
prognostic
SEPs were also divided 2=N20
The
CH 69.2%;
of mortality with type II was 76% (III
100%~). The mortality
in all pathologies.
aetiological
N. Barficrini, F’. Giannini,
(Institute
for Nervous and Mental
Disease, University
of Sienna, Italy)
within the first or second day following the
of survival after 3 months following by Mjoen
(XI’s).
the prognostic value of each modality and in the different
event leading to brain damage. proposed
middle latency
potentials
identifiable
in 3
unilaterally;
There was 56% mortality associated
In order to evaluate the neuropathic carbamazepine studied
(CBZ)
10 patients
generalised neurological
(6
M,
during At
41’ mean
signs. A neurophysiological NCVs
from
before starting the treatment
which
therapeutic
and central
of
nervous systems we
age 32.4
years)
affected
by
convulsive seizures in the absence of an organic lesion or
limbs, sensory and motor performed
effects of chronic administration
on peripheral
CBZ
blood
examination upper
(SEPs from upper
and lower
limbs) was
and after 18 months of therapy,
concentration
was
maintained
within
range.
follow-up
several
neurophysiotogical
parameters
showed
a