The electrocardiogram in exercise

The electrocardiogram in exercise

SELECTFD 2 501 ABSTIiACTS and a mean of 0.08 second. The author concludes that a QRS less than 0.08 is normal; 0.09 to 0.10 may be normal; over 0.1...

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SELECTFD 2

501

ABSTIiACTS

and a mean of 0.08 second. The author concludes that a QRS less than 0.08 is normal; 0.09 to 0.10 may be normal; over 0.10 is usually evidence of prolonged Two hundred and eighty-seven other electrocardiaintraventricular conduction. grams were analyzed in which S-T in Lead I or II or in both was depressed and these compared with normals. It was found that while the scatter was toward the longer duration of QRS,, 1practically all QRS durations fell in the normal range. KATZ.

Herve, L. Luis, and cise. Rev. argent.

Santander, Manuel Besoain: de eardiol. 6: 299, 1939.

The

Electrocardiogram

in Exer-

The alterations of the electrocardiogram caused by exercise were investigated Small variations in the amplitude in thirty normal subjects o.f different ages. of the waves and in the duration of intervals between them were found as well as depression of the S-T interval of even more than 2 mm. in some cases. In cardiac patients pathologic alterations appear after exercise in 27 per cent of the cases xhieh had normal electrocardiograms at rest; this proportion is even higher (35 per cent) if only those patients with coronary disease are considered. The alterations induced by exercise and considered indicating myocardial disease of coronary origin are: inversion of the T wave in any of the leads in which it was positive, appearance of arrhythmia or auriculoventrieular or intraventricular block, and prolongation of the Q-T intexXval. The depression value as a sign

of

of the S-T interval coronary insufficiency.

induced

by

exercise

has

a very

restricted AUTHORS.

Kang,

Ole:

Qonorrhoeal

Myocarditis.

Brit.

1\I. J. 1: 117,

1940.

Apart from the myocardial involvement in the bacterial endoearditis of gonococcal septicemia, myocarditis is seldom established as a complication in gonococcal infection. In view of this it seems surprising that a series of six instances of what is regarded as gonorrhea1 myacarditis was encountered in the course of one year in sn ordinary medical ward. It seems probable that this condition often escapes detection, since suggestive symptoms may be absent. The diagnosis was made on the ground that all six cases had changes in the electrocardiogram occurring in the course of acute nr chronic complicated gonococcal infection. In some instances the myocarditis was slight and transitory, as may be the case in many simple infections, for example, pneumonia. However, in two cases incomplete bundle branch block developed and persisted, and in a third case the angina1 pain still troubled the patient considerably when he was reexamined a year later. Such a course suggests the possibility that some of these patients will return to hospital sooner or later with chronic heart disease. It is possible, also, that some of the chronic (‘degenerative” heart diseases which we see today arise from gonococeal infection. The gonococcal complement-fixation reaction has been instrumental in pointing out how great a proportion of so-called rheumatic arthritis cases were really gonorrheal. It may be worth while to search the anamnesis of heart patients for gonorrhea as we search it for syphilis and rheumatic fever, and the complementfixation reaction may revea.1 that some instances of heart lesion attributed to rheumatic infection ought to be regarded &s following on persistent gonococcal infection. AUTHOR.