- Email: [email protected]
The systolic murmur in aortic stenosis
SELECTED Malinow, M. Exe&sing
R., Moia, Iscbemic
B., Otero, Muscle.
Q89
ABSTRACTS
E., and Garcia I. Preliminary
A.:
Circulatory Observatious.
Changes
Produced
by
Rev. argent. de cardiol.
15:l (Feb.), 1948. Standardized weight-lifting exercises (lifting of five-pound weights forty times per minute over 5.0 cm. for three minutes) were performed on five normal subjects. A slight increase in heart rate and in mean arterial pressure was noted. The pressure changes reversed themselves regularly and a fall in systolic and diastolic pressures occurred on recovery. Exercising of an ischemic limb resulted in a slightly higher rise of arterial pressure with a lessened tendency to an overswing of pressure values below the resting value. The heart rate increased as before. Atropine and ergonovine did not greatly alter the response obtained. The studies are preliminary to similar observations in patients with intermittent claudication and to studies on the pathogenesis of pain arising from an ischemic extremity. HECH1. Del
Zar, L. E., and Bronstein, J.: The Iduemce of Infiltration cordial Region on tbe Occurrence of Precordial Pain and Changea in An&na Pectoris. Rev. argent. de cardiol. 15:17
on
Aneatbesia of the Eleetroeardiographic
Pre-
(Feb.), 1948.
Twenty or 40 mg. of 1 per cent procaine, given subcutaneously in the precordial area, prevented or attentuated the occurrence of precordial pain in seven of ten patients subjected to an changes on exertion were not altered exercise test. In eighteen patients the electrocardiographic by the procedure, even when pain was obliterated. A full explanation of the discrepancy is not given, but it appears unlikely that precordial procaine infiltration altered coronary blood flow, as has been suggested before. HECAT. Alzamora-Castro, Aortie Stenosis.
V.,
Rubio,
C.
W.,
and
Battihma,
G.
D.:
The
Systolic
Murmur
in
Rev. argent. de cardiol. 15:25 (Feb.), 1948.
Simultaneous registration of arterial pulse curves and phonocardiograms reveal that the systolic murmur of aortic and pulmonic stenosis begins at the onset of the ejection period and fades toward the latter part of systole. The maximal intensity of the murmur coincides with the maximal ejection period. HECHT. Etala,
F., and Syndrome.
J. A.: Anatomical-Clinical Study of a Case of Post-tachycardial Rev. argent. de cardiol. 15:133 (Feb.), 1948.
Berreta,
Cossio, Gonzllex-&bath%, Berconsky, and Vedoya, in three successive articles, have described a clinical picture which they called “post-tachycardial syndrome,” based on the following data: (a) relatively young subjects, usually without demonstrable cardiac lesions; (b) repeated and prolonged attacks of paroxysmal tachycardia, mostly ventricular; (c) reversible cardiac enlargement; (d) electrocardiographic changes after the attack consisting of depression of the RS-T segment in one lead with elevation in another, inversion of the T wave, and prolongation of the Q-T interval; (e) gradual return of the electrocardiograph to normal within a few days; and, (f) at post mortem, the presence of a dilated and hypertrophied myocardium with absence of other lesions. The authors report an additional case with post-mortem study. The histologic study failed to reveal any typical change in the myocardium or in the coronary system. Even if the death in the reported case cannot be attributed with certainty to this syndrome, the latter cannot be considered as completely benign because it may be followed by sudden death or by heart failure. LUISADA.
R., Moia, Iscbemic
B., Otero, Muscle.
Q89
ABSTRACTS
E., and Garcia I. Preliminary
A.:
Circulatory Observatious.
Changes
Produced
by
Rev. argent. de cardiol.
15:l (Feb.), 1948. Standardized weight-lifting exercises (lifting of five-pound weights forty times per minute over 5.0 cm. for three minutes) were performed on five normal subjects. A slight increase in heart rate and in mean arterial pressure was noted. The pressure changes reversed themselves regularly and a fall in systolic and diastolic pressures occurred on recovery. Exercising of an ischemic limb resulted in a slightly higher rise of arterial pressure with a lessened tendency to an overswing of pressure values below the resting value. The heart rate increased as before. Atropine and ergonovine did not greatly alter the response obtained. The studies are preliminary to similar observations in patients with intermittent claudication and to studies on the pathogenesis of pain arising from an ischemic extremity. HECH1. Del
Zar, L. E., and Bronstein, J.: The Iduemce of Infiltration cordial Region on tbe Occurrence of Precordial Pain and Changea in An&na Pectoris. Rev. argent. de cardiol. 15:17
on
Aneatbesia of the Eleetroeardiographic
Pre-
(Feb.), 1948.
Twenty or 40 mg. of 1 per cent procaine, given subcutaneously in the precordial area, prevented or attentuated the occurrence of precordial pain in seven of ten patients subjected to an changes on exertion were not altered exercise test. In eighteen patients the electrocardiographic by the procedure, even when pain was obliterated. A full explanation of the discrepancy is not given, but it appears unlikely that precordial procaine infiltration altered coronary blood flow, as has been suggested before. HECAT. Alzamora-Castro, Aortie Stenosis.
V.,
Rubio,
C.
W.,
and
Battihma,
G.
D.:
The
Systolic
Murmur
in
Rev. argent. de cardiol. 15:25 (Feb.), 1948.
Simultaneous registration of arterial pulse curves and phonocardiograms reveal that the systolic murmur of aortic and pulmonic stenosis begins at the onset of the ejection period and fades toward the latter part of systole. The maximal intensity of the murmur coincides with the maximal ejection period. HECHT. Etala,
F., and Syndrome.
J. A.: Anatomical-Clinical Study of a Case of Post-tachycardial Rev. argent. de cardiol. 15:133 (Feb.), 1948.
Berreta,
Cossio, Gonzllex-&bath%, Berconsky, and Vedoya, in three successive articles, have described a clinical picture which they called “post-tachycardial syndrome,” based on the following data: (a) relatively young subjects, usually without demonstrable cardiac lesions; (b) repeated and prolonged attacks of paroxysmal tachycardia, mostly ventricular; (c) reversible cardiac enlargement; (d) electrocardiographic changes after the attack consisting of depression of the RS-T segment in one lead with elevation in another, inversion of the T wave, and prolongation of the Q-T interval; (e) gradual return of the electrocardiograph to normal within a few days; and, (f) at post mortem, the presence of a dilated and hypertrophied myocardium with absence of other lesions. The authors report an additional case with post-mortem study. The histologic study failed to reveal any typical change in the myocardium or in the coronary system. Even if the death in the reported case cannot be attributed with certainty to this syndrome, the latter cannot be considered as completely benign because it may be followed by sudden death or by heart failure. LUISADA.