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Transactivation Of EGFR1 By Urokinase Requires The Co-receptor EGFR4
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ASSOCIATION FOR ACADEMIC SURGERY AND SOCIETY OF UNIVERSITY SURGEONS—ABSTRACTS
59.6. A Novel Method Of Inducing Abdominal Aortic Aneurysms In Rodents. A. Laser,1 G. Lu,1 A. Ghosh,1 K. Roelofs,1 B. McEvoy,1 P. DiMusto,1 C. M. Bhamidipati,2 G. Ailawadi,2 J. L. Eliason,1 P. K. Henke,1 G. R. Upchurch1,2; 1University of Michigan, Ann Arbor, MI; 2University of Virginia, Charlottesville, VA
59.7. The Mechanism Of Diabetes Pathophysiology Diminishes AAA Formation: Glucose Influences AGE-induced MMP-9 Expression. G. Banker, S. Marsh, F. Zhang, J. Lengfeld, K. Kent, B. Liu; Department of Surgery, University of Wisconsin School of Medicine, Madison, Wisconsin
59.8. Angiotensin Receptor Blockers Suppress Abdominal Aortic Aneurysm (AAA) In Angiotensin II-induced ApoE Knockout Mice. Y. Iida,1 G. M. Schultz,1 M. M. Dua,1 N. Miyama,1 B. Xu,1 S. Rea Peterson,2 S. Sulaimon,2 R. L. Dalman1; 1Stanford University School of Medicine, Stanford, CA; 2Medtronic Vascular Inc., Santa Rosa, CA
59.9. Effectiveness Of A Novel Limited-Spectrum Matrix Metalloprotease Inhibitor On Model Aneurysms. T. Ennis, J. Jin, B. Arif, J. A. Curci; Washington University School of Medicine, St. Louis, MO
59.10. Membrane Type 1-Matrix Metalloproteinase As A Possible Mediator Of Estrogen-Stimulated Matrix Degradation And Migration In Vascular Smooth Muscle Cells. D. J. Mountain, S. S. Kirkpatrick, D. C. Cassada, S. L. Stevens, M. B. Freeman, M. H. Goldman, O. H. Grandas; University of Tennessee Graduate School of Medicine, Knoxville, TN 59.11. Effect Of Metabolic Syndrome On The Response To Arterial Injury. Y. Fu, E. A. Duru, K. A. Youker, M. G. Davies; Department of Cardiovascular Surgery, Methodist DeBakey Heart and Vascular Center, Houston, TX 59.12. Changes In MMP2 System Following Arterial Injury In Metabolic Syndrome. Y. Fu, E. A. Duru, Z. Chen, M. G. Davies; Department of Cardiovascular Surgery, Methodist DeBakey Heart and Vascular Center, Houston, TX
59.13. Changes In UPA Following Arterial Injury In Metabolic Syndrome. Y. Fu, E. A. Duru, Z. Chen, M. G. Davies; Department of Cardiovascular Surgery, Methodist DeBakey Heart and Vascular Center, Houston, TX
59.14. Differential Modulation Of Proteases And Protease Inhibitors Expression In In Vitro Metabolic Syndrome. E. A. Duru, Y. Fu, M. G. Davies; The Methodist Hospital Research Institute, Houston, TX
59.15. Modulation Of Leptin, Adiponectin, And Resistin Expression In In Vitro Metabolic Syndrome. E. A. Duru, Y. Fu, M. G. Davies; Methodist Hospital Research Institute, Houston, TX
59.16. Modulation Of Inflammatory Markers By S-1-P In In Vitro Metabolic Syndrome. E. A. Duru, Y. Fu, M. G. Davies; Methodist Hospital Research Institute, Houston, TX
59.17. Novel Adipokine Resistin Augments Injury-Induced Intimal Hyperplasia In A Murine Model. R. Ding1; 1 Department of Surgery, Stanford University, Stanford, California; 2Monta Vista High School, Cupertino, California; 3 Changhai Hospital, Shanghai, Shanghai; 4Department of Molecular Environmental Biology, University of California, Berkley, Berkley, California
59.18. Adipocytokine Resistin Increases Endothelial Monolayer Permeability In Human Coronary Artery Endothelial Cells. M. Jamaluddin, L. Zhang, Q. Yao, C. Chen; Baylor College of Medicine, Houston, TX
VASCULAR 3: ENDOTHELIAL CELL DYSFUNCTION 60.1. Pharmacologic Pre-conditioning With The Gasotransmitter Hydrogen Sulfide Occurs Through The Xanthine Oxidase Pathway. N. Jimenez, P. W. Henderson, A. J. Reiffel, Y. H. Millet, J. A. Spector; Weill Cornell Medical College, New York, New York
60.2. Urokinase Requires NAD(P)H Oxidase To Transactivate EGFR. M. G. Davies, E. A. Duru, Y. Fu; Methodist DeBakey Heart and Vascular Center, Houston, TX
60.3. Transactivation Of EGFR1 By Urokinase Requires The Co-receptor EGFR4. M. G. Davies, Y. Fu, E. A. Duru; Methodist DeBakey Heart and Vascular Center, Houston, TX
60.4. Ginsenoside Rb1 Reverses HomocysteineInduced Endothelial Dysfunction In Human Coronary Artery Endothelial Cells. L. Zhang,2 R. Ding,1 S. Powell,1 W. Zhou1; 1 Department of Surgery, Division of Vascular and Endovascular Surgery, Stanford University, Stanf, Stanford, California; 2 Department of Vascular Surgery, Changhai Hospital, Second Military Medical University, Shanghai, Shanghai, Shanghai
60.5. Synergistic Effect Of Disturbed Flow With Chemical Stimulation By Thrombin On Tissue Factor Expression In HUVEC. R. Abe B. E. Sumpio; Yale University School of Medicine, New Haven, CT
60.6. Pomegranate Extract Blocks Ritonavir-Induced Endothelial Dysfunction In Porcine Pulmonary Arteries. S. M. Weakley, T. J. Beadle, C. Cheng, X. Wang, J. Jiang, P. Kougias, P. H. Lin, Q. Yao, C. Chen; Baylor College of Medicine, Houston, TX
ASSOCIATION FOR ACADEMIC SURGERY AND SOCIETY OF UNIVERSITY SURGEONS—ABSTRACTS
59.6. A Novel Method Of Inducing Abdominal Aortic Aneurysms In Rodents. A. Laser,1 G. Lu,1 A. Ghosh,1 K. Roelofs,1 B. McEvoy,1 P. DiMusto,1 C. M. Bhamidipati,2 G. Ailawadi,2 J. L. Eliason,1 P. K. Henke,1 G. R. Upchurch1,2; 1University of Michigan, Ann Arbor, MI; 2University of Virginia, Charlottesville, VA
59.7. The Mechanism Of Diabetes Pathophysiology Diminishes AAA Formation: Glucose Influences AGE-induced MMP-9 Expression. G. Banker, S. Marsh, F. Zhang, J. Lengfeld, K. Kent, B. Liu; Department of Surgery, University of Wisconsin School of Medicine, Madison, Wisconsin
59.8. Angiotensin Receptor Blockers Suppress Abdominal Aortic Aneurysm (AAA) In Angiotensin II-induced ApoE Knockout Mice. Y. Iida,1 G. M. Schultz,1 M. M. Dua,1 N. Miyama,1 B. Xu,1 S. Rea Peterson,2 S. Sulaimon,2 R. L. Dalman1; 1Stanford University School of Medicine, Stanford, CA; 2Medtronic Vascular Inc., Santa Rosa, CA
59.9. Effectiveness Of A Novel Limited-Spectrum Matrix Metalloprotease Inhibitor On Model Aneurysms. T. Ennis, J. Jin, B. Arif, J. A. Curci; Washington University School of Medicine, St. Louis, MO
59.10. Membrane Type 1-Matrix Metalloproteinase As A Possible Mediator Of Estrogen-Stimulated Matrix Degradation And Migration In Vascular Smooth Muscle Cells. D. J. Mountain, S. S. Kirkpatrick, D. C. Cassada, S. L. Stevens, M. B. Freeman, M. H. Goldman, O. H. Grandas; University of Tennessee Graduate School of Medicine, Knoxville, TN 59.11. Effect Of Metabolic Syndrome On The Response To Arterial Injury. Y. Fu, E. A. Duru, K. A. Youker, M. G. Davies; Department of Cardiovascular Surgery, Methodist DeBakey Heart and Vascular Center, Houston, TX 59.12. Changes In MMP2 System Following Arterial Injury In Metabolic Syndrome. Y. Fu, E. A. Duru, Z. Chen, M. G. Davies; Department of Cardiovascular Surgery, Methodist DeBakey Heart and Vascular Center, Houston, TX
59.13. Changes In UPA Following Arterial Injury In Metabolic Syndrome. Y. Fu, E. A. Duru, Z. Chen, M. G. Davies; Department of Cardiovascular Surgery, Methodist DeBakey Heart and Vascular Center, Houston, TX
59.14. Differential Modulation Of Proteases And Protease Inhibitors Expression In In Vitro Metabolic Syndrome. E. A. Duru, Y. Fu, M. G. Davies; The Methodist Hospital Research Institute, Houston, TX
59.15. Modulation Of Leptin, Adiponectin, And Resistin Expression In In Vitro Metabolic Syndrome. E. A. Duru, Y. Fu, M. G. Davies; Methodist Hospital Research Institute, Houston, TX
59.16. Modulation Of Inflammatory Markers By S-1-P In In Vitro Metabolic Syndrome. E. A. Duru, Y. Fu, M. G. Davies; Methodist Hospital Research Institute, Houston, TX
59.17. Novel Adipokine Resistin Augments Injury-Induced Intimal Hyperplasia In A Murine Model. R. Ding1; 1 Department of Surgery, Stanford University, Stanford, California; 2Monta Vista High School, Cupertino, California; 3 Changhai Hospital, Shanghai, Shanghai; 4Department of Molecular Environmental Biology, University of California, Berkley, Berkley, California
59.18. Adipocytokine Resistin Increases Endothelial Monolayer Permeability In Human Coronary Artery Endothelial Cells. M. Jamaluddin, L. Zhang, Q. Yao, C. Chen; Baylor College of Medicine, Houston, TX
VASCULAR 3: ENDOTHELIAL CELL DYSFUNCTION 60.1. Pharmacologic Pre-conditioning With The Gasotransmitter Hydrogen Sulfide Occurs Through The Xanthine Oxidase Pathway. N. Jimenez, P. W. Henderson, A. J. Reiffel, Y. H. Millet, J. A. Spector; Weill Cornell Medical College, New York, New York
60.2. Urokinase Requires NAD(P)H Oxidase To Transactivate EGFR. M. G. Davies, E. A. Duru, Y. Fu; Methodist DeBakey Heart and Vascular Center, Houston, TX
60.3. Transactivation Of EGFR1 By Urokinase Requires The Co-receptor EGFR4. M. G. Davies, Y. Fu, E. A. Duru; Methodist DeBakey Heart and Vascular Center, Houston, TX
60.4. Ginsenoside Rb1 Reverses HomocysteineInduced Endothelial Dysfunction In Human Coronary Artery Endothelial Cells. L. Zhang,2 R. Ding,1 S. Powell,1 W. Zhou1; 1 Department of Surgery, Division of Vascular and Endovascular Surgery, Stanford University, Stanf, Stanford, California; 2 Department of Vascular Surgery, Changhai Hospital, Second Military Medical University, Shanghai, Shanghai, Shanghai
60.5. Synergistic Effect Of Disturbed Flow With Chemical Stimulation By Thrombin On Tissue Factor Expression In HUVEC. R. Abe B. E. Sumpio; Yale University School of Medicine, New Haven, CT
60.6. Pomegranate Extract Blocks Ritonavir-Induced Endothelial Dysfunction In Porcine Pulmonary Arteries. S. M. Weakley, T. J. Beadle, C. Cheng, X. Wang, J. Jiang, P. Kougias, P. H. Lin, Q. Yao, C. Chen; Baylor College of Medicine, Houston, TX