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Traumatic vasospastic dystrophy of the extremities
SELECTED
Atlas, Lawrence N.: Surg.
42:
1042,
285
ABSTRACTS
Traumatic Vasospastic Dystrophy of the Extremities.
.A&.
1941.
Trauma to an extremity with injury to a blood vessel or its nerve supply may result in a syndrome which the author calls traumatic vasospastic dystrophy. AS a result of trauma to a blood vessel, without ischemia, there may occur pain, subjective and objective coldness, hyperhidrosis, cyanosis, tense, shiny skin, firm, tender edema, muscular weakness, joint fixation, and decalcification of bone. Three patients with this syndrome are presented. Theories as to the cause of the clinical picture are discussed. NAIDE.
Roome, N. W.: M.
A. J. 44:
Sympathetic 594, 1941.
Blockade
in
Peripheral Vascular
Accidents.
Canad.
Blockade of the lumbar sympathetic chain, by infiltration with procain solution, was found to give prompt and complete relief of otherwise intractable pain in two cases of embolism of the lower extremities. The method is recommended for trial in cases of embolism, either alone or in combination with embolectomy, and should also be considered in the management of wounds of the major arteries, and of arterial thrombosis and acute thrombophlebitis. AUTHOR. Master, A. M.: J. Roentgenol.
Roentgenoscopy 45:
350,
as
a Diagnostic Aid in Coronary Occlusion. Am.
1941.
Roentgenoscopic observations of ventricular contraction in 300 private patients are recorded. More than half of these had coronary occlusion; the remainder included other types of heart disease and normal subjects. The technique employed is described in detail. Roentgenoscopy is shown to be a simple and inexpensive method of diagnosing coronary occlusion with myocardial infarction. Abnormalities in pulsation were present in 75 per cent of the cases of coronary occlusion. Systolic expansion (reversal of pulsation) of the left ventricle, observed in 50 per cent of these cases, is characteristic, if not pathognomonic, of myocardial infarction. ‘ ‘ Lag ’ ’ and ( ( doubling ’ ’ of pulsation are incomplete forms of systolic expansion. Systolic expansion is seen in practically every case of large heart with ventricular aneurysm. Absence and diminution of pulsation were present in 25 per cent of cases with coronary occlusion, but a,lso occurred in other types of heart disease. The abnormal pulsations observed were located in the apical and the supra-apical portions of the left ventricle in almost 85 per cent of the cases studied. The evidence of abnormal contraction did not depend upon the location of the infarct as determined electrocardiographically. The incidence of abnormal pulsation in coronary occlusion was found to be greater when the heart was enlarged. When the area of abnormal pulsation was large, the prognosis was poor. Systolic expansion may appear directly after the coronary occlusion and persist for many years. Its disappearance, or a change to absence or diminution of pulsation, is of favorable significance. In normal hearts the pulsations are always normal. Hypertension alone does not produce changes. Disease of the coronary artery, without occlusion, did not exhibit systolic expansion or absence of pulsation. When these conditions are
Atlas, Lawrence N.: Surg.
42:
1042,
285
ABSTRACTS
Traumatic Vasospastic Dystrophy of the Extremities.
.A&.
1941.
Trauma to an extremity with injury to a blood vessel or its nerve supply may result in a syndrome which the author calls traumatic vasospastic dystrophy. AS a result of trauma to a blood vessel, without ischemia, there may occur pain, subjective and objective coldness, hyperhidrosis, cyanosis, tense, shiny skin, firm, tender edema, muscular weakness, joint fixation, and decalcification of bone. Three patients with this syndrome are presented. Theories as to the cause of the clinical picture are discussed. NAIDE.
Roome, N. W.: M.
A. J. 44:
Sympathetic 594, 1941.
Blockade
in
Peripheral Vascular
Accidents.
Canad.
Blockade of the lumbar sympathetic chain, by infiltration with procain solution, was found to give prompt and complete relief of otherwise intractable pain in two cases of embolism of the lower extremities. The method is recommended for trial in cases of embolism, either alone or in combination with embolectomy, and should also be considered in the management of wounds of the major arteries, and of arterial thrombosis and acute thrombophlebitis. AUTHOR. Master, A. M.: J. Roentgenol.
Roentgenoscopy 45:
350,
as
a Diagnostic Aid in Coronary Occlusion. Am.
1941.
Roentgenoscopic observations of ventricular contraction in 300 private patients are recorded. More than half of these had coronary occlusion; the remainder included other types of heart disease and normal subjects. The technique employed is described in detail. Roentgenoscopy is shown to be a simple and inexpensive method of diagnosing coronary occlusion with myocardial infarction. Abnormalities in pulsation were present in 75 per cent of the cases of coronary occlusion. Systolic expansion (reversal of pulsation) of the left ventricle, observed in 50 per cent of these cases, is characteristic, if not pathognomonic, of myocardial infarction. ‘ ‘ Lag ’ ’ and ( ( doubling ’ ’ of pulsation are incomplete forms of systolic expansion. Systolic expansion is seen in practically every case of large heart with ventricular aneurysm. Absence and diminution of pulsation were present in 25 per cent of cases with coronary occlusion, but a,lso occurred in other types of heart disease. The abnormal pulsations observed were located in the apical and the supra-apical portions of the left ventricle in almost 85 per cent of the cases studied. The evidence of abnormal contraction did not depend upon the location of the infarct as determined electrocardiographically. The incidence of abnormal pulsation in coronary occlusion was found to be greater when the heart was enlarged. When the area of abnormal pulsation was large, the prognosis was poor. Systolic expansion may appear directly after the coronary occlusion and persist for many years. Its disappearance, or a change to absence or diminution of pulsation, is of favorable significance. In normal hearts the pulsations are always normal. Hypertension alone does not produce changes. Disease of the coronary artery, without occlusion, did not exhibit systolic expansion or absence of pulsation. When these conditions are