Abstracts

372

Birchall

Journal of the American Academy of Dermatology

et a l

9. Adzick NS, Khan SH, Bondoc CC, Quinby WC, Remensnyder JP. Management of toxic epidermal necrolysis in ~a pediatric burn center. Am J Dis Child 1985; 139:499-502. 10. Artz CP, Rittenbury MS, Yarborough DR. An appraisal of allografts and xenografts as biological dressings for wounds and bums. Ann Surg 1972;175:934-5. 11. Monafo WW, Chuntrasakul C, Ayrazian V. Hypertonic sodium solutions in the treatment of bum shock. Am J Surg 1973; 126:778-83. 12. Arstikaitis MJ. Ocular aftermath of Stevens-Johnson syndrome. Arch Ophthalmol 1973;90:376-9. 13. Smith DA, Burgdoff WHC. Universal cutaneous depig-

14.

~5.

16. 17.

mentation following phcnytoin-induced toxic epidermal necrolysis. J AM ACAD DERMATOL 1984;10:106-9. Klumlovsky FA, Delgreco F, Herdson PB, Lazar P. Renal disease associated with toxic epidermal necroiysis (Lyell's disease). Am J Med 1974;57:817-25. Stenn KS, Madri JA, Roll FJ. Migrating epidermis produces AB2 collagen and requires continual collagen synthesis for movement. Nature 1979;277:229-32. Medawar PB. The cultivation of adult mammalian skin epithelium in vitro. Q J Micro Sci 1948;89:187-96. Westly ED, Wechsler HL. Toxic epidermal necrolysis: granulocytic leukopenia as a prognostic indicator. Arch Dermatol 1984;120:721-6.

ABSTRACTS

Morphoea (localised scleroderma) in a patient with mixed connective tissue disease Golding DN. Ann Rheum Dis 1986;45:523-5

Late metastases of cutaneous malignant melanoma Steiner A, Wolf C, Pehamberger H, Wolff K. Br J Dermatol 1986;114:737-40

Morphea in an 18-year-old girl may or may not be related to her mixed connective tissue disease. Mixed connective tissue disease has a speckled pattern antinuclear antibody, negative deoxyribonucleic acid binding, negative Sm antibodies, and a very high anti-ribonucleuprotein titer. No other features of scleroderraa were found. Philip C. Anderson, M.D.

Late metastasis (after 10 years disease-free) is more commor~than the accumulated literature would suggest, No therapy is known in any event, Philip C. Anderson, M,D,

Chemotactic lipoxygenase products in sera from patients with psoriasis

Androphy EJ. Ciba Found Syrup 1986;120:221-34

Fogh K, Ternowitz T, Kragballe K, Herlin T. Arch Dermatol Res 1986;278:173-6 Another expensive study compares blood chemistry in psoriatic patients only with disease-free people. By now everyone wonders why comparably "skin-diseased" corttrols are not customary. Again, we find that persons sick with psoriasis differ from wholly healthy people, this time in regard to some arachidonate metabolites. Philip C. Anderson, M.D.

Anti-oxidant effects of retinoids on inflammatory skin diseases Yoshioka A, Miyachi Y, Irnamura S, N i w a Y. Arch Dermatol Res 1986;278:177-83 By a number of different measurements it seems that retinoids have an antioxidative action. Philip C. Anderson, M.D.

Exacerbation of pemphigus by rifampicin Miyagawa S, Yamashina Y, Okuchi T, Konoike Y, Kano T, Sakamoto K. Br J Dermatol 1986;114:729-32 Rifampicin seemed plainly to aggravate this patient's psoriasis, probably indirectly, by interfering with the steroid medication. Philip C. Anderson, M.D.

Papillomaviruses and interferon

The use of interferons is back in the mainstream of interest. A review is given of the mechanisms for papillomavirus control. Philip C. Anderson, M.D.

lmmunohistoehemieal demonstration o[ hyalinosis-assoeiated 90kD glyeoprotein in amyloid deposits of lichen amyloidosus Teppo AM, Kariniemi AL, Maury CP. Am J Clin Pathol 1986;86:175-9 Lichen arnyloldosus may be a different sort of disorder than the nodular types, biochemically. The lichenoid form contains the glycoprotein found in hair follicles, termed "90kD glycoprotein." Amyloid disorders are quite heterogenous as regards their chemistry. Philip C. AtMerson, M.D.

Buccai cellulitis reevaluated Chartrand SA, Harrison CJ. Am J Dis Child 1986;140: 891-3 Buceal cellulitis in infants is serious, requiring prompt therapy. The median age was 11 months in this sample of seventy-two cases. Fifty-five percent of all patients are dangerously bacteremic early. Hemophilus hTfluenzae is the most common cause, but indistinguishable cases may be due to other organisms, according to these pediatricians from the University of Oklahoma. Philip C. Anderson, M.D.